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Epigenetic silencing of microRNA-137 enhances ASCT2 expression and tumor glutamine metabolism

During the preparation of this work, the author(s) used ChatGPT to correct the grammar, language and used Google Gemini to create Figure 1

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The redox activity of hemoglobins: from physiologic functions to pathologic mechanisms

Specifically, as proposed in a hypothetical model shown in Figure 7(b), if a protein or a small molecule target could be designed under diabetic conditions to reduce metabolic pressure on complex I, that is, relaying excess electrons from NADH to CoQ, such a target could serve as a potential therapeutic approach by restoring NADH/NAD + redox balance in the absence of enhanced proton pumping and ROS production