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Neuropsychopharmacology 1994;10:129-38

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Adduct formation of AcH with Cys or cysteinylglycine rather than GSH has also been suggested as a mechanism of EtOH-induced GSH depletion [50], however, the steady state levels of these thiols are much lower that for GSH in the liver

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10.1101/gad.908001 Fernandez AM, Dupont J, Farrar RP, Lee S, Stannard B, Le Roith D: Muscle-specific inactivation of the IGF-I receptor induces compensatory hyperplasia in skeletal muscle