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Stronger effects observed in older adult populations may reflect unique pathophysiological mechanisms of late-life depression, including mitochondrial dysfunction and neurodegenerative processes, which ALC could plausibly target

FIGURE 6 Epigallocatechin-3-gallate nanoparticles suppressed the protein expression of mitogen-activated protein kinases signaling pathways after atopic dermatitis In order to explore the signaling pathways-mediated necroptosis after AD, the p-p38, ERK1, and ERK2 were detected by immunohistochemistry

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Clinical response to aminoglycoside therapy: importance of the ratio of peak concentration to minimal inhibitory concentration