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doi: 10.1016/j.wneu.2015.09.052

Mild ferroptosis is proposed to impair repair (190), but it is unknown whether there is a threshold of ferroptosis that switches repair from adaptive to maladaptive, or which molecular signals govern this switch

Introduction Ovarian cancer (OC), a serious obstetrical and gynecological malignant disease, ranks eighth in terms of morbidity and mortality overall, resulting in huge economic and health problems ( Glutathione S-transferase Mu (GSTM) gene family is group of 5 proteins, GSTM1-5, that play a key role in the detoxification of electrophilic compounds, such as cancer-causing toxins, anticarcinogens and products of oxidative stress via conjugating with glutathione ( a of the sulfhydryl group of reduced glutathione (GSH) from 9.0 in aqueous solution to about 6.5 when GSH is bound in the active site ( For clearly elucidate GSTM gene family in OC, especially in prognostic and expression significance

The vial goes cloudy, the peptide aggregates, and the dose is non-functional

Thnh phn: Lm sng da: Vi thnh phn cha phc hp Glutathione gip lm sng da, m thm nm cho ln da