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The hippocampus, critical for memory processing, is particularly vulnerable to A-induced damage, leading to memory deficits commonly observed in AD ( Another important factor in AD is the dysregulation of homeostasis in calcium levels

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While this gap precludes claims of therapeutic readiness, the consistency of antimicrobial, anti-virulence, and host-modulatory effects across diverse preclinical models provides a strong rationale for biomarker-driven early-phase studies designed explicitly around infectious contexts