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Furthermore, pharmacological and/or genetic approaches implicate both actin [65,67,6972] and microtubules ([7376] and reviewed in [77]) in insulin-stimulated GLUT4 traffic

Attention to reproductive endpoints and long-term outcomes should anchor this agenda so that Erastin-class approaches can be tested credibly for clinical benefit in EMS

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39 ASCT1 and ASCT2, which have mainly been studied in the context of glutamine dependence, are expressed at high levels in different cancer types, 40,41,42 prompting these transporters to be considered putative therapeutic targets in cancer, with different inhibitors currently under investigation