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Mitochondrial dysfunction is not only the result of oxidative stress and inflammatory responses but also a trigger for selective autophagy (mitophagy)

But shifting your injection day by 2-3 days one week creates a peak-trough pattern that amplifies side effects

The first study identified N -acetyl-aspartyl-glutamate (NAAG), which can be hydrolyzed to glutamate when needed by the oncogenic cells via glutamate carboxypeptidase II (GCP II) [41, 42], suggesting that GCPII is a viable target for cancer therapy, either alone or in combination with glutaminase inhibition

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Rossello Aubach L, Fornos Roca X, Fernandez Alvarez ME