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In the cybrid cells, treatment or overexpression of MOTS-c was able to increase mitochondrial complex subunits (ubiquinol-cytochrome c reductase, complex III

Through the halogenation or peroxidase cycle, MPO could generate reactive oxygen species (ROS) effectively [36]

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The results of this study suggested that DLL3 had potential to be a viable clinical target, despite manageable toxicity noted