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The acquired cysteines cause decreased stability or impaired DNA binding of the tumor suppressor p53, while KRAS oncogenes are constitutively activated

Recent progress in transcriptomics, proteomics, and single-cell technologies is beginning to illuminate the heterogeneity and context-specific plasticity of astrocyte subtypes across disease states, offering an unprecedented opportunity to develop targeted interventions with cell-type and disease-stage selectivity

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Phosphorylated transcription factors may enter the nucleus and potentially modulate the transcription of specific genes that have been associated with growth hormone synthesis and secretion

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