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It is worth noting that in addition to the strategies of directly intervening in metabolic pathways mentioned above, Enalapril, an angiotensin-converting enzyme inhibitor (ACE-I), represents a new method of reducing plasma TMAO levels, although the underlying mechanism has not been discovered (Konop et al., 2018)

Our findings suggest the dose used was insufficient to elicit a robust response in all participants, but with this dose, the incidence of adverse events was relatively low and was consistent with toxicities observed with other AAV therapies (including those using other liver-targeting capsids)

Doppler tissue evaluation of atrial conduction properties in patients with non-alcoholic fatty-liver disease

A.ChenW.JingX.et al (2023)

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