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Ammonium-evoked alterations in intracellular sodium and pH reduce glial glutamate transport activity

Epub 2012 Dec 5

FTH neuron-specific knockout (FTH-KO) mice are more susceptible to ferroptosis after TBI, and the absence of FTH offsets the protective effect of melatonin on ferroptosis induced by TBI, 659 suggesting that melatonin plays a protective role at least in part by inhibiting FTH-mediated ferroptosis in neurons after TBI

lymphoma versus pancreatic ductal adenocarcinoma (PDAC)], exerting distinct pressures on T cell metabolism and function and precluding a blanket therapeutic approach (143)

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