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KDM5B knockdown in breast cancer cells activated the AMPK protein, reduced the level of lipid metabolism, and inhibited the epithelial-mesenchymal transition (EMT), and reduced their growth and migration ability, revealing novel theoretical evidence for the clinical regulation of tumor metabolism by targeting related genes

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In serous ovarian cancer (SOC), ALOX5AP is highly expressed and is associated with poor prognosis and aggressive disease features, such as lymphatic invasion, resistance to platinum chemotherapy, and poor survival rates