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Lack of improvement after six months may indicate need for protocol adjustment or additional interventions

Rather than manipulating individual pathways in isolation, effective intervention will likely require coordinated, context-dependent modulation of chaperones, autophagy, UPS activity, and ER stress responses

In conditions where renal blood flow is dependent upon prostaglandin synthesis, administration of NSAIDs can result in significant decreases in renal blood flow leading to acute renal failure

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While these approaches can be helpful, they rarely lead to lasting improvement if the primary pain mechanism is not clearly identified and treated