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10.1016/j.cmet.2011.07.001 Cell Metab

184, No

Haploinsufficiency of the SLC52A1 (OMIM#615026) due to maternal microdeletion and heterozygous intronic variant has been reported to cause a transient riboflavin responsive neonatal multiple acyl-CoA dehydrogenase deficiency that resolved with oral supplementation of riboflavin [27-29]