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Here, we propose that nMFBs function essentially as trolling lines within the large intestinal nuclei, scavenging inappropriate materials from the nuclear interior and moving them to the periphery for removal (Fig 16B)

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232 During breast cancer progression, the stiffened fibrotic tumor microenvironment enhances the TGF autocrine pathway in tumor-associated macrophages (TAMs), activating their collagen biosynthesis program

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