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& Pertea, M

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Upon CCCP treatment, Miro interacting with wild-type LRRK2 was degraded with time, while in the LRRK2 G2019S mutant, Miro showed decreased interaction with the mutant as well as resistance to degradation, delaying mitochondrial arrest and clearance in iPSC-derived neurons (Singh et al., 2019)

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Moreover, short- and long-term tunicamycin treatments that trigger ER stress via UDP-HexNAc inhibition were ineffective against ferroptosis (Supplementary Fig

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Media high 257 Forman HJ, Zhang H, Rinna A (2009) Glutathione: Overview of its protective roles, measurement, and biosynthesis

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Nutritional and metabolic factors in amyotrophic lateral sclerosis

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