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Samples with different expression pattern are highlighted (yellow)

Conversely, cuproptosis, a copper-induced cell death triggered by the accumulation of Cu in mitochondria, leads to the aggregation of lipoylated dihydrolipoamide S-acetyltransferase (DLAT) and a loss of Fe-S protein, which are associated with energy formation in the tricarboxylic acid cycle (TCA), leading to proteotoxic cellular stress, eventually leading to cell death [7]

& Vande Velde, C

The key steps include: 1) Formation of Acyl-CoA : Fatty acids are activated in the cytosol by combining with Coenzyme A (CoA), forming acyl-CoA

Moreover, L-Carnitine supplementation has been suggested to prevent protein catabolism [47], which is corroborated by the increase in mTOR expression observed by the end of the study