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Nature 239 , 395397 (1972)

In an animal model of high-salt diet-induced hypertensive nephropathy, iron accumulation was observed in injured kidneys, accompanied by renal inflammation, mitochondrial dysfunction, oxidative stress, massive proteinuria, and sustained intravascular hemolysis [257, 258]

This dual actionoptimizing current energy production and expanding overall mitochondrial capacityis crucial for patients whose cellular batteries have been depleted by chronic viral persistence or long-term systemic inflammation

Loss of Grx1 significantly relieved the lung damage caused by hyperoxia (Fig

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