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Through Gene Ontology (GO) analysis, the researchers demonstrated that these differentially expressed proteins were mainly related to innate immune responses, cytokine expression, and apoptosis

Trpm2 deficiency in microglia attenuates neuroinflammation during epileptogensis by upregulating autophagy via the AMPK/mTOR pathway

Complex II ROS Complex II, also known as succinate dehydrogenase (SDH) or succinate coenzyme Q reductase (SQR), is recognized as a significant source of ROS in mammalian mitochondria

Proteomic and secretomic comparison of young and aged dermal fibroblasts highlights cytoskeleton as a key component during aging

[5] The third theory suggests that restricted venous drainage from the brain may be impaired resulting in congestion