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therefore, if we know what the mechanisms involved in glial and neuronal alterations are, we must be able to identify the elements that can be used as effector elements and design drugs to control and reduced harm during the stage of relapse [14]

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ROS induce hypermethylation of the Runt-related transcription factor 3 (RUNX3) gene promoter region, which correlates with increased plasma protein carbonylation and is associated with a higher risk of HBV-related HCC [214]