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[DOI] [PMC free article] [PubMed] [Google Scholar] 86.Loomba R, Neutel J, Mohseni R, Bernard D, Severance R, Dao M, Saini S, Margaritescu C, Homer K, Tran B, et al

Mercury treatment further depletes the mitochon- drial enzymes, in turn, leading to severe mitochondrial damage as documented in our earlier reports leading to cellular metabolic insult in cells and tissue [65,66]

The atherogenicity of Lp(a) is substantially greater than that of LDL on a per-particle basis, and elevated Lp(a) has been established as an independent risk factor for ASCVD, particularly in individuals with high LDL-C

Reactive oxygen species-induced autophagic degradation of Helicobacter pylori CagA is specifically suppressed in cancer stem-like cells