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At the molecular level, zinc deficiency downregulates TJ proteins such as claudin-3 and occludin, leading to increased permeability and compromised barrier integrity (Zhong et al., 2010)

The markedly reduced selenoprotein status observed in inflammatory rheumatic musculoskeletal diseases supports the rationale for controlled interventional studies using selenium supplementation, particularly in patients with confirmed selenium deficiency

Here, NOX2-derived O 2 .- is released into phagocytotic vesicles to kill internalised pathogens through respiratory bursts 89

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