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The purpose of this article is to clarify and explain the significant role of the oxidative stress in PD pathogenesis

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Our results show that health sedentary individuals already show significant cellular and bioenergetic downregulations compared to moderately active individuals which could, over time, lead to the development of multiple NCDs

Interestingly this mutation is located in a residue next to the LNCaP prostate cancer cell line mutation Thr878Ala (139, 225), supporting the view that this region in the ligand binding domain of the androgen receptor is very susceptible to mutagenesis in prostate cancer, which may lead to the tumor becoming resistant to hormone-based therapies