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The long-term stabilization of synaptic changes is determined by a parallel increase of pre- and postsynaptic structures such as axonal bouton, dendritic spine and postsynaptic density

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Namely, in analogy, we evidenced the occluded superior sagittal sinus-produced central disturbances (i.e., intracranial (superior sagittal sinus) hypertension) rapidly transmitted to the periphery (i.e., portal, caval hypertension, aortal hypotension, multiorgan lesions) [20]

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