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TLR signal transduction transpires through two primary channels: the myeloid differentiation primary response 88 (MYD-88)-independent and MYD-88-dependent pathways, which engage a consortium of adaptor proteins that facilitate the propagation of activation signals and amplify pro-inflammatory responses (Ashayeri Ahmadabad et al

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Micro-exon genes MEGs were predicted as previously described 9 with further manual refinement using available S

The -SH group of Cys25 participates in the formation of acylenzyme intermediate with the substrate (shown in black)