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139 Initially, it was hypothesized that the loss or reduction of ABCG2-mediated renal urate secretion would result in increased renal urate reabsorption, as diminished renal excretion is typically considered the primary mechanism of hyperuricemia in most gout patients

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doi: 10.1007/s00253-018-8789-8 137

In summary, vitamin K, via VKORC1L1, quenches lipid peroxides, prevents ferroptosis, and interrupts an NF-B/TNF inflammatory circuit, thereby promoting endothelial repair

Nuclear magnetic resonance metabolomic profiling of urine provides a noninvasive alternative to the identification of biomarkers associated with endometriosis