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The loss of the chondrocytic phenotype and proliferative potential during in vitro expansion, termed as dedifferentiation, has been well documented

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Impaired insulin-stimulated glucose transport (insulin resistance) is thought to be due to reduced GLUT4 delivery to the cell surface, which has been measured in both human muscle and adipose tissue [4,5]

The transfer of heme via an intermediate compartment would minimise the release of cytotoxic heme into the cytoplasm and facilitate the regulated movement of heme (Severance and Hamza 2009)

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