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3H), essentially mirroring the partial effects on the transcriptome observed upon the re-introduction of IF1 (Fig

In all, 32 individuals had a complete clinical response and 17 had a complete pathological response

doi: 10.1007/s00403-024-02872-x

All these findings suggest that GSTP1 overexpression, beyond its role in drug resistance, may contribute to the metastatic ability of cancers including promoting EMT, modulating MMPs, and upregulating certain signaling pathways, thus becoming an attractive target for metastasis research