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This is thought to involve both the entry of extracellular Ca2+ via plasma membrane receptor operated Ca2+ channels and the release of Ca2+ from intracellular Ca2+ storages, particularly in the endoplasmicreticulum (calciosome).129 Cytosolic Ca2+ elevation is a major determinant of the oxidative injury.130 As stated above, Ca2+ facilitates mitochondrial permeability transition, a key event leading to both necrosis and apoptosis

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(2022) investigated the insecticidal potential of 40 indigenous plant species, including Portulaca oleracea L., on Asian citrus psyllids ( Diaphorina citri )

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