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Using mouse embryonic fibroblasts (MEFs) and human malignant melanoma cells treated with active-site mTOR inhibitor (asTORi), was demonstrate that mTORC1 stimulates the translation of MTFP1 mediated by 4E-BP, and therefore the mTOR inhibition induces the phosphorylation of the DRP1 at Ser 637, this phosphorylation prevents it translocation to mitochondria, conversely, the pro-fission phosphorylation site of DRP1 at Ser 616 was decreased in asTORi treated cells

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The peroxisomes, microsomes, and mitochondria in parenchymal cells lead the production of ROS by regulating the PPAR, which is involved in the expression of genes related to fatty acid oxidation

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A p-value of less than 0.05 was considered statistically significant