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During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes

Building on previous findings, the current research aimed to assess the protective roles of BCA, CoQ10, and PHL against oxidative stress, nephritic apoptosis, and molecular disruptions caused by simultaneous chromium and arsenic exposure in Swiss albino mice

Increased Expression of System Xc- in Glioblastoma Confers an Altered Metabolic State and Temozolomide Resistance

PD and DLB are the most studied diseases of -syn at present [28, 29]

[DOI] [PubMed] [Google Scholar] 2.Newsholme P, Lima MM, Procopio J, Pithon-Curi TC, Doi SQ, Bazotte RB, et al