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This review examines the molecular mechanisms underlying ASM-associated oxidative stress in clinical, non-kindled/kindled preclinical, as well as non-epilepsy models, and highlights evidence demonstrating the ability of ascorbic acid to attenuate ROS accumulation, enhance endogenous antioxidant defenses, and modulate redox-sensitive signaling pathways implicated in epileptogenesis and ASM-induced toxicity

255,256,257 Liver-specific deletion of Lgr4 significantly restrains liver regeneration

[DOI] [PubMed] [Google Scholar] 124.Keith B., Johnson R.S., Simon M.C

This powerful antioxidant therapy helps protect brain cells from oxidative stress and supports cognitive function, providing relief from symptoms and enhancing quality of life

Statements Author contributions YK: Supervision, Writing original draft, Conceptualization