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PDXs closely mimic human tumors and preserve genetic and physical traits in a three-dimensional setting, but they are expensive, time-consuming, and may eventually lose their human tissue components

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Accumulating evidence links such mitochondrial remodeling in females to oxidative stress and altered neuronal excitability as mechanisms through which diverse early-life stressors become biologically embedded, leading to sustained changes in cellular energetics and circuit function [68, 69, 70]

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The others have compelling anecdotal data and decent mechanistic logic, but far fewer robust human trials for fat loss specifically