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In a portion of PINK1 mutants, expression of human PINK1 in the Drosophila testes restores male fertility and normal mitochondrial morphology (Clark et al., Numerous studies indicate that mitochondrial pathology and muscle and dopaminergic degeneration due to Drosophila PINK1 inactivation and PINK1 mutant can be rescued by PRKN overexpression and downregulation of Miro (Yang et al., 2006

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224 However, when damage exceeds the capacity of MQC system, stress-induced mPTP opening and MOMP can release mtDNA or other DAMPs into the cytoplasm, triggering inflammation and cell death

Ducks had been suggested as reservoir of O