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However, N-terminal modifications, such as N-acetylation, D-norleucine substitution, or replacement with -aminobutyric acid (GABA), dramatically elongated the peptides half-lives

Progesterone attenuates Abeta25-35-induced neuronal toxicity via JNK inactivation and progesterone receptor membrane component 1-dependent inhibition of mitochondrial apoptotic pathway

Transcriptome profiling reveals infection strategy of an insect maculavirus

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Altered responsiveness to extracellular ATP enhances acetaminophen hepatotoxicity