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TB-500 operates systemically as a **cytoskeleton remodeler**: its primary function is G-actin monomer sequestration (active N-terminal Ac-Ser-Asp-Lys-Pro), its cleaved AcSDKP sequence is an **autonomous angiogenic factor**, it upregulates laminin-5 and matrix metalloproteinases (MMPs), and mobilizes mesenchymal progenitor cells

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Heischmann S, Gano LB, Quinn K, Liang LP, Klepacki J, Christians U, et al

However, there is currently some controversy about G-CSF therapy, and its efficacy and safety need to be verified through more clinical trials