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That dual approval reflects the compound mechanism of action, which treats weight and metabolic health as interconnected problems rather than separate issues

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Its dual agonist mechanism simultaneously activates both the GIP receptor (GIPR) and the GLP-1 receptor (GLP-1R), producing synergistic effects on glucose homeostasis, appetite regulation, and adipose tissue metabolism that exceed those achievable through either pathway alone [1]

During oocyte development from the GV to M phase, MAMs serve as initiation sites for mitochondrial division