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This three layer device, consisting of a fibrin hydrogel cell layer, a mechanical actuation layer, and a glass cover, recapitulated joint dynamics and showed that SYN321 downregulated inflammatory markers, reduced matrix metalloproteinase activity, limited extracellular matrix degradation, and preserved microtissue architecture (Palma et al., 2024)

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PMID: 83 TsayJJVelezACollazoDLaniadoIBessichJMurthyVet al

Observations of neurodevelopmental or neurodegenerative disease in humans, triggered by mutations in various proteins involved in autophagy, provide additional support for this hypothesis, including those affecting PINK1, Parkin, OPTN, TBK1, ATL1, SQSTM1, and WDR81, as well as ATG5, AP4, HTT, WIPI4, and DYNC1H1 (Zhu et al