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Nuclear DNA damage after acetaminophen overdose is likely to be caused by DNase(s) unrelated to the caspase-activated DNase, which is typically responsible for DNA fragmentation during apoptosis

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Although research on the mechanisms of tumor energy metabolism has been ongoing for several decades and efforts have been made to treat tumors through targeting energy metabolism, the current challenges persist, leading to limited therapeutic outcomes

However, this method has been shown to be of limited clinical value since a considerable amount of guanine may originate from non-house-dust mite sources (Hallas et al., 1993)

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