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doi: 10.1016/j.redox.2015.09.009 94 KrstiJTrivanoviDMojsiloviSSantibanezJF

Although disparate, common features of these treatments have led to a novel hypothesis involving a role of glucotoxicity, the polyol pathway, and induction of the glutathione synthetic enzyme, GCL, in the mechanism of neuroprotection in SCI

Molecular mechanism of the short-term cardiotoxicity caused by 2,3-dideoxycytidine (ddC): modulation of reactive oxygen species levels and ADP-ribosylation reactions

These treatments help clients uncover the root causes of their addiction and address both the physical and psychological aspects of their addiction

2005;63:S475S476