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Clinical Implications The study concludes by suggesting potential treatment avenues

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Chang et al., 2015) linked the peroxiredoxin-1 (PRX-1) status in glioma to OS caused by therapy and showed that a decreased level of PRX-1 was associated with a better response to chemotherapy (Dittmann et al., 2011) and thioredoxin (TRX) (Saitoh et al., 1998), when bound to apoptosis signal-regulating kinase 1 (ASK1), inhibited their phosphorylation and the subsequent activation of the cell death pathways that follow their dissociation under OS

Certainly, emerging therapeutic intervention strategies for regulating ROSincluding gene therapy, stem cell therapy, and combination therapies such as antioxidant chemotherapyhave already demonstrated promising applicational potential

Differential regulation of CD4(+) T helper cell responses by curcumin in experimental autoimmune encephalomyelitis