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Similarly, in lipopolysaccharide (LPS)-induced cardiomyocytes, METTL3 deficiency inhibited LPS-induced ferroptosis by upregulating the expression of SLC7A11 in an m 6 A methylation-dependent manner[160]

Their morphology and functions vary depending on developmental stage, CNS region, species, sex, and physiological or pathological context (Fig

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Oxidative stress is another critical contributor to AD pathogenesis

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