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This review attempts to provide some mechanistic insight (Figures 13) into how the bi-directional network between the gut microbiota and the brain can not only protect but also trigger and aid chronic gut inflammation, promote a global pro-inflammatory state, modify and contribute to protein misfolding processes, dysregulate the autophagic-lysosomal protein clearance mechanism, differentiate T-cell subsets to facilitate a deleterious autoimmune response, and finally modulate immune recruitment and BBB status

Effects of NT-814, a dual neurokinin 1 and 3 receptor antagonist, on vasomotor symptoms in postmenopausal women: a placebo-controlled, randomized trial

Oxidative stress and vascular damage are considered two major cellular factors that lead to alterations in the TM

The recombinant pMut1-sacB-gmR and pMut2-sacB-ampR plasmids were constructed using NEBuilder HiFi DNA Assembly Master Mix (New England Biolabs) according to the manufacturers instructions and authenticated via whole plasmid sequencing

Mitochondrial respiration deficits driven by reactive oxygen species in experimental temporal lobe epilepsy