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These multi-targeted agents highlight that effective cardioprotection in MIRI may require simultaneously addressing multiple pathological pathways, including oxidative stress, inflammation, and metabolic dysfunction

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Inhibiting NNMT with 5-Amino-1MQ restores NAD+ pools, which activates AMPK (AMP-activated protein kinase), the cellular energy sensor that shifts metabolism from glucose storage to fat oxidation

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However, it is important to note that not all of these indicators are exclusive to endothelium dysfunction