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Therapeutic strategies that enhance antioxidant capacity (e.g., hyperbaric oxygen[74]), modulate metabolism and inflammation (e.g., adenosine[75]), or reduce cardiac workload (e.g., atenolol[72]) have shown protective potential, highlighting the importance of targeting metabolic instability and oxidative damage

Patients with complex chronic illnesses frequently experience unpredictable symptom flares and newly developed food intolerances

Despite the above-mentioned discrepancy, it appears that GHRs are central to the regulation of the quinone redox state, likely preventing toxicity of quinones, either naturally present or found as environmental pollutants

In pursuit of alleviating cell damage caused by glutamate-induced excitotoxicity, current drug research mainly focuses on NMDAR antagonists