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The SIRT3 deficiency triggers a decline in renal function, a significant increase in inducible nitric oxide synthase (iNOS), and upregulation of the expressions of toll-like receptor 4 (TLR4), NF-B, inhibitor of Kappa B (IB), inhibitor of kappa B kinase (IKK), and p65 proteins

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In other words, high-quality males would have more robust immune function were it not for the immunosuppressive effects of elevated testosterone.) This previous work suggests that although testosterone is a key mediator of investment in males sexual development and mating behavior [1012], it may not be directly responsible for decrements in immunity that occur in the context of elevated reproductive effort

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