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Further research is warranted to determine exact mechanisms of radioprotection, which may help us better identify radioprotective agents

Indeed, Orai3 protein, in comparison to Orai1, lacks the homolog of cysteine 195 residue which is reactive to oxidation (Santn-Mrquez et al., 2019), resulting in the reduced expression of proinflammatory proteins (Park et al., 2017)

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Set7/9 directly interacts with p65/RelA and modulates NFkB-dependent transcription As an alternative to the H3K4-methylation-dependent mechanism that mediates up-regulation of p65/RelA expression, Li et al

Nature 274 (5674), 923924 (1978)