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On the mechanistic level [6], investigations have expanded beyond traditional core pathways such as OS, mitochondrial dysfunction, and neuroinflammation to encompass complex networks involving multi-organ system interactions like the gut-brain axis and spleen-brain axis

These findings led us to explore the consequences of PUFA exposure on IECs with impaired GPX4 activity in the intestine

pylori (Kouitcheu Mabeku et al

Pharmacological modulation of nitric oxide release: new pharmacological perspectives, potential benefits and risks

Disulfiram, a clinically used anti-alcoholism drug and copper-binding agent, induces apoptotic cell death in breast cancer cultures and xenografts via inhibition of the proteasome activity