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During all stages of atherogenesis, endothelial dysfunction, oxidative stress and inflammation contribute to disease development and progression (Nus and Mallat, 2016)

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This programmed inflammatory cell death, orchestrated by the caspase superfamily, plays a pivotal role in pulmonary injury via canonical (e.g., caspase-1) or non-canonical pathways (Yu et al., 2021)

Urine acylglycine concentrations were further corrected for renal function and expressed as mmol/mol creatinine (crt)

These findings establish innate RNA sensing as an intrinsic driver of hematopoietic stem cell aging and highlight modulation of MDA5-dependent pathways as a potential strategy to mitigate age-related hematopoietic dysfunction