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(2021) 404(Pt B):124050

DTI), [2] and drug screening assays, such as stem cell-derived hepatocyte-like cells, that are capable of detecting toxicity early in the drug development process

2e), implying that the cnd1-K658E mutation is sufficient to cause chromosome segregation defects without affecting cell viability at a detectable level

Consequently, the development of therapeutic agents that disrupt various aspects of Gln metabolism may represent a promising anticancer strategy ( Figure 2 )

Renal dysfunction not only affects traditional renal markers but also alters metabolites such as phosphate, uric acid, creatinine, and urea